Scientific Research On Cellular Nutrients in Fighting CancerClinical Studies on Cellular Nutrients Megadose vitamins in bladder cancer: a double-blind clinical trial. L-arginine stimulates host defenses in patients with breast cancer. A population-based case-control study of carotenoid and vitamin A intake and ovarian cancer (United States). Influence of drinking green tea on breast cancer malignancy among Japanese patients. Dietary antioxidant intake and the risk of cardia cancer and noncardia cancer of the intestinal and diffuse types: a population-based case-control study in Sweden Dietary carotenoids and vitamins A, C, and E and risk of breast cancer. A new concept of tumor promotion by tumor necrosis factor-alpha, and cancer preventive agents (-)-epigallocatechin gallate and green tea--a review. Two stages of cancer prevention with green tea. Cancer inhibition by green tea. Dietary catechins and cancer incidence among postmenopausal women: the Iowa Women's Health Study (United States). Severe hypovitaminosis C in lung-cancer patients: the utilization of vitamin C in surgical repair and lymphocyte-related host resistance. Ascorbic acid in the plasma and blood cells of women with breast cancer. The effect of the consumption of food with an elevated content of this vitamin Endogenous urinary 3-hydroxyproline has 96% specificity and 44% sensitivity for cancer screening. Effects of selenium and zinc supplementation on nutritional status in patients with cancer of digestive tract. The relationship of dietary selenium and breast cancer. Reduction of cancer mortality and incidence by selenium supplementation. Urinary tea polyphenols in relation to gastric and esophageal cancers: a prospective study of men in Shanghai, China. Research Data on Cellular Nutrients Anticlastogenic, antigenotoxic and apoptotic activity of epigallocatechin gallate: a green tea polyphenol. Decreased incidence of prostate cancer with selenium supplementation: results of a double-blind cancer prevention trial. Growth inhibition, cell-cycle dysregulation, and induction of apoptosis by green tea constituent (-)-epigallocatechin-3-gallate in androgen-sensitive and androgen-insensitive human prostate carcinoma cells. Induction of apoptosis by green tea catechins in human prostate cancer DU145 cells. Effect of black and green tea polyphenols on c-jun phosphorylation and H(2)O(2) production in transformed and non-transformed human bronchial cell lines: possible mechanisms of cell growth inhibition and apoptosis induction. Inhibition of bcl-x(l) phosphorylation by tea polyphenols or epigallocatechin-3-gallate is associated with prostate cancer cell apoptosis. Induction of p57 is required for cell survival when exposed to green tea polyphenols. Green tea polyphenol epigallocatechin-3 gallate induces apoptosis of proliferating vascular smooth muscle cells via activation of p53. Selenium effects on prostate cell growth. P53 gene of chang-liver cells (Atcc-Ccl13) exposed to aflatoxin B1 (Afb): the effect of lysine on mutation at codon 249 of exon 7. Success of L-lysine therapy in frequently recurrent herpes simplex infection. Treatment and prophylaxis. Amino acid deficiency up-regulates specific mRNAs in murine embryonic cells. Identification and characterization of amino acid starvation-induced CD24 gene in MCF-7 human breast cancer cells. Retinoids and differentiation treatment: a strategy for treatment in cancer. Vitamins and cancer. Anticarcinogenic effects of (-)-epigallocatechin gallate. Mechanistic aspects of green tea as a cancer preventive: effect of components on human stomach cancer cell lines. Inhibition of lung metastasis in mice induced by B16F10 melanoma cells by polyphenolic compounds. Cell signaling and regulators of cell cycle as molecular targets for prostate cancer prevention by dietary agents. Association of suppression of extracellular signal-regulated kinase phosphorylation by epigallocatechin gallate with the reduction of matrix metalloproteinase activities in human fibrosarcoma HT1080 cells. Inhibition of urinary bladder tumors induced by N-butyl-N-(4-hydroxybutyl)-nitrosamine in rats by green tea. Inhibitory effects of tea catechins, black tea extract and oolong tea extract on hepatocarcinogenesis in rat. Chemoprevention of mammary tumor virus-induced and chemical carcinogen-induced rodent mammary tumors by natural plant products. Effects of catechins on the mouse lung carcinoma cell adhesion to the endothelial cells. Tea polyphenols down-regulate the expression of the androgen receptor in LNCaP prostate cancer cells. Induction of apoptosis and cell cycle arrest in cancer cells by in vivo metabolites of teas. Green tea polyphenol (-)-epigallocatechin 3-gallate inhibits MMP-2 secretion and MT1-MMP-driven migration in glioblastoma cells. (-)-Epigallocatechin gallate, the main constituent of Japanese green tea, inhibits tumor promotion of okadaic acid Anti-metastatic activity of curcumin and catechin. Growth inhibition of leukemic cells by (-)-epigallocatechin gallate, the main constituent of green tea Chemopreventive effects of green tea polyphenols correlate with reversible induction of p57 expression Inhibitory effect of green tea extract on the process of pancreatic carcinogenesis induced by N-nitrosobis-(2-oxypropyl)amine (BOP) and on tumor promotion after transplantation of N-nitrosobis-(2-hydroxypropyl)amine (BHP)-induced pancreatic cancer in Syrian hamsters Inhibition of collagenases from mouse lung carcinoma cells by green tea catechins and black tea theaflavins Tumor gelatinases and invasion inhibited by the green tea flavanol epigallocatechin-3-gallate Inhibitory effects of green tea catechins on the activity of human matrix metalloproteinase 7 (matrilysin) Tea catechins and related polyphenols as anti-cancer agents Inhibition of fibroblast growth factors by green tea Green tea catechins and vitamin E inhibit angiogenesis of human microvascular endothelial cells through suppression of IL-8 production Green tea polyphenols induce apoptosis in vitro in peripheral blood T lymphocytes of adult T-cell leukemia patients The specific anti-cancer activity of green tea (-)-epigallocatechin-3-gallate (EGCG). Tea catechin synergies in inhibition of cancer cell proliferation and of a cancer specific cell surface oxidase (ECTO-NOX) Involvement of protein tyrosine phosphorylation in the effect of green tea polyphenols on Ehrlich ascites tumor cells in vitro. Potent inhibitory action of red wine polyphenols on human breast cancer cells. Green tea: cancer preventive beverage and/or drug. Green tea polyphenol epigallocatechin-3 gallate inhibits Her-2/neu signaling, proliferation, and transformed phenotype of breast cancer cells. Green tea (Camellia sinensis) extract and its possible role in the prevention of cancer. Cell cycle dysregulation by green tea polyphenol epigallocatechin-3-gallate. Green tea polyphenol epigallocatechin-3-gallate differentially modulates nuclear factor kappaB in cancer cells versus normal cells. Green Tea Constituent (-)-Epigallocatechin-3-Gallate Inhibits Hep G2 Cell Proliferation and Induces Apoptosis through p53-Dependent and Fas-Mediated Pathways [In Process Citation] Role of the retinoblastoma (pRb)-E2F/DP pathway in cancer chemopreventive effects of green tea polyphenol epigallocatechin-3-gallate. Matrix metalloproteinase inhibition by green tea catechins. Induction of apoptosis in prostate cancer cell lines by the green tea component, (-)-epigallocatechin-3-gallate. Induction of apoptosis by the green tea flavonol (-)-epigallocatechin-3-gallate in human endothelial ECV 304 cells. Green tea extracts decrease carcinogen-induced mammary tumor burden in rats and rate of breast cancer cell proliferation in culture. Cancer chemoprevention by tea polyphenols through mitotic signal transduction blockade. Effects of epigallocatechin-3-gallate on growth, epidermal growth factor receptor signaling pathways, gene expression, and chemosensitivity in human head and neck squamous cell carcinoma cell lines. Green tea polyphenol targets the mitochondria in tumor cells inducing caspase 3-dependent apoptosis. Involvement of caspase-3 in epigallocatechin-3-gallate-mediated apoptosis of human chondrosarcoma cells. Epigallocatechin gallate protects U937 cells against nitric oxide-induced cell cycle arrest and apoptosis. Green tea polyphenol epigallocatechin-3 gallate induces apoptosis of proliferating vascular smooth muscle cells via activation of p53. Protective effects of green tea polyphenols and their major component, (-)-epigallocatechin-3-gallate (EGCG), on 6-hydroxydopamine-induced apoptosis in PC12 cells. Effect of vitamin C on prostate cancer cells in vitro: effect on cell number, viability, and DNA synthesis. Effect of vitamin C on androgen independent prostate cancer cells (PC3 and Mat-Ly-Lu) in vitro: involvement of reactive oxygen species-effect on cell number, viability and DNA synthesis. Combined vitamins B12b and C induce the glutathione depletion and the death of epidermoid human larynx carcinoma cells HEp-2. Antimetastatic and anti-invasive ability of phospho-ascorbyl palmitate through intracellular ascorbate enrichment and the resultant antioxidant action Inhibitory effect of dietary arginine on growth of Ehrlich ascites tumor cells in mice. Dietary copper and dimethylhydrazine affect protein kinase C isozyme protein and mRNA expression and the formation of aberrant crypts in colon of rats. (-)-Epigallocatechin-3-gallate in Camellia sinensis leaves from Himalayan region of Sikkim: inhibitory effects against biochemical events and tumor initiation in Sencar mouse skin. Detrimental effect of cancer preventive phytochemicals silymarin, genistein and epigallocatechin 3-gallate on epigenetic events in human prostate carcinoma DU145 cells. Green tea and its catechins inhibit breast cancer xenografts. (-)-Epigallocatechin gallate can prevent cisplatin-induced lung tumorigenesis in A/J mice. (-)-Epigallocatechin (EGC) of green tea induces apoptosis of human breast cancer cells but not of their normal counterparts. Gene expression profile in human prostate LNCaP cancer cells by (--) epigallocatechin-3-gallate. Green tea and prostate cancer. Epigallocathechin-3 gallate selectively inhibits the PDGF-BB-induced intracellular signaling transduction pathway in vascular smooth muscle cells and inhibits transformation of sis-transfected NIH 3T3 fibroblasts and human glioblastoma cells (A172). Inhibitory effects of green tea infusion on in vitro invasion and in vivo metastasis of mouse lung carcinoma cells. Inhibition of tumour invasion and angiogenesis by epigallocatechin gallate (EGCG), a major component of green tea. Novel approaches to chemoprevention of skin cancer. A major constituent of green tea, EGCG, inhibits the growth of a human cervical cancer cell line, CaSki cells, through apoptosis, G(1) arrest, and regulation of gene expression. Effect of tea polyphenols on growth of oral squamous carcinoma cells in vitro. Role of p53 and NF-kappaB in epigallocatechin-3-gallate-induced apoptosis of LNCaP cells. Molecular pathway for (-)-epigallocatechin-3-gallate-induced cell cycle arrest and apoptosis of human prostate carcinoma cells. Mechanisms of growth inhibition of human lung cancer cell line, PC-9, by tea polyphenols. Inhibition of bladder tumor growth by the green tea derivative epigallocatechin-3-gallate. Blocking telomerase by dietary polyphenols is a major mechanism for limiting the growth of human cancer cells in vitro and in vivo. Chemoprevention of oral cancer by green tea. Green tea and cancer chemoprevention. Growth inhibition and regression of human prostate and breast tumors in athymic mice by tea epigallocatechin gallate. Inhibitory effects of (-)-epigallocatechin gallate on spontaneous hepatoma in C3H/HeNCrj mice and human hepatoma-derived PLC/PRF/5 cells. Anticarcinogenic activity of green tea polyphenols. Inhibition of skin tumor promoter-caused induction of epidermal ornithine decarboxylase in SENCAR mice by polyphenolic fraction isolated from green tea and its individual epicatechin derivatives. Progress in studies on the antimutagenicity and anticarcinogenicity of green tea epicatechins. Molecular targets for green tea in prostate cancer prevention. Suppression of human pancreatic carcinoma cell growth and invasion by epigallocatechin-3-gallate. Lung cancer prevention with (-)-epigallocatechin gallate using monitoring by heterogeneous nuclear ribonucleoprotein B1. Biochemical changes in the connective tissue components of malignant tumors and lungs in mice during metastatic spreading and chemotherapy Differential effects and transport kinetics of ascorbate derivatives in leukemic cell lines. Tea polyphenols: prevention of cancer and optimizing health Activation mechanisms of matrix metalloproteinases. Inhibitory effect of six green tea catechins and caffeine on the growth of four selected human tumor cell lines. Inhibition of growth and induction of apoptosis in human cancer cell lines by tea polyphenols. Epigallocatechin-3-gallate-induced stress signals in HT-29 human colon adenocarcinoma cells. Suppression of extracellular signals and cell proliferation through EGF receptor binding by (-)-epigallocatechin gallate in human A431 epidermoid carcinoma cells. Matrix metalloproteinase inhibition by green tea catechins. Green tea constituent epigallocatechin-3-gallate and induction of apoptosis and cell cycle arrest in human carcinoma cells. Anti-metastatic effect of an autooxidation-resistant and lipophilic ascorbic acid derivative through inhibition of tumor invasion Growth suppression of malignant leukemia cell line in vitro by ascorbic acid (vitamin C) and its derivatives. Ascorbic acid (vitamin C) improves the antineoplastic activity of doxorubicin, cisplatin, and paclitaxel in human breast carcinoma cells in vitro. Selenium-induced inhibition of angiogenesis in mammary cancer at chemopreventive levels of intake. Inhibition of aminopeptidase N (AP-N) and urokinase-type plasminogen activator (uPA) by zinc suppresses the invasion activity in human urological cancer cells. Novel role of zinc in the regulation of prostate citrate metabolism and its implications in prostate cancer. Esophageal cancer prevention in zinc-deficient rats: rapid induction of apoptosis by replenishing zinc. Induction of apoptosis and necrosis by zinc in human thyroid cancer cell lines. Effect of zinc on immune functions and host resistance against infection and tumor challenge. Zinc inhibits nuclear factor-kappa B activation and sensitizes prostate cancer cells to cytotoxic agents. The effect of trace elements--especially zinc--on metastasizing of Lewis lung tumor Selenium inhibition of N-methyl-N-nitrosourea-induced mammary carcinogenesis in the rat. |
Page Tools:
What's New:
- The latest attempt of the drug lobby to discredit the health benefits of vitamins
- Victory over Cancer!
- Dismantling the ‘Brussels EU’ – a precondition for global natural health freedom
- Brussels EU continues attempts to curtail spread of lifesaving natural health information
- How to use diagnostic technologies wisely and for the benefit of your health
- Radio interview – Jon Rappoport Show
- President Obama, Wake Up America!
- Wake Up, America! Prevent Dicatorship And War!
- What the 'Brussels EU' isn't telling you about the Lisbon Treaty
- The Oil Cartel's Goal: Toppling the Obama Government
